The **brms** package comes with a lot of built-in response distributions – usually called *families* in R – to specify among others linear, count data, survival, response times, or ordinal models (see `help(brmsfamily)`

for an overview). Despite supporting over two dozen families, there is still a long list of distributions, which are not natively supported. The present vignette will explain how to specify such *custom families* in **brms**. By doing that, users can benefit from the modeling flexibility and post-processing options of **brms** even when using self-defined response distributions.

As a case study, we will use the `cbpp`

data of the **lme4** package, which describes the development of the CBPP disease of cattle in Africa. The data set contains four variables: `period`

(the time period), `herd`

(a factor identifying the cattle herd), `incidence`

(number of new disease cases for a given herd and time period), as well as `size`

(the herd size at the beginning of a given time period).

```
herd incidence size period
1 1 2 14 1
2 1 3 12 2
3 1 4 9 3
4 1 0 5 4
5 2 3 22 1
6 2 1 18 2
```

In a first step, we will be predicting `incidence`

using a simple binomial model, which will serve as our baseline model. For observed number of events \(y\) (`incidence`

in our case) and total number of trials \(T\) (`size`

), the probability mass function of the binomial distribution is defined as

\[ P(y | T, p) = \binom{T}{y} p^{y} (1 - p)^{N-y} \]

where \(p\) is the event probability. In the classical binomial model, we will directly predict \(p\) on the logit-scale, which means that for each observation \(i\) we compute the success probability \(p_i\) as

\[ p_i = \frac{\exp(\eta_i)}{1 + \exp(\eta_i)} \]

where \(\eta_i\) is the linear predictor term of observation \(i\) (see `vignette("brms_overview")`

for more details on linear predictors in **brms**). Predicting `incidence`

by `period`

and a varying intercept of `herd`

is straight forward in **brms**:

In the summary output, we see that the incidence probability varies substantially over herds, but reduces over the cource of the time as indicated by the negative coefficients of `period`

.

```
Family: binomial
Links: mu = logit
Formula: incidence | trials(size) ~ period + (1 | herd)
Data: cbpp (Number of observations: 56)
Samples: 4 chains, each with iter = 2000; warmup = 1000; thin = 1;
total post-warmup samples = 4000
Group-Level Effects:
~herd (Number of levels: 15)
Estimate Est.Error l-95% CI u-95% CI Rhat Bulk_ESS Tail_ESS
sd(Intercept) 0.75 0.23 0.39 1.28 1.00 1233 1854
Population-Level Effects:
Estimate Est.Error l-95% CI u-95% CI Rhat Bulk_ESS Tail_ESS
Intercept -1.40 0.26 -1.91 -0.89 1.00 2209 2087
period2 -1.00 0.31 -1.61 -0.41 1.00 4552 2908
period3 -1.15 0.33 -1.80 -0.53 1.00 4656 3139
period4 -1.62 0.45 -2.57 -0.76 1.00 4714 2802
Samples were drawn using sampling(NUTS). For each parameter, Bulk_ESS
and Tail_ESS are effective sample size measures, and Rhat is the potential
scale reduction factor on split chains (at convergence, Rhat = 1).
```

A drawback of the binomial model is that – after taking into account the linear predictor – its variance is fixed to \(\text{Var}(y_i) = T_i p_i (1 - p_i)\). All variance exceeding this value cannot be not taken into account by the model. There are multiple ways of dealing with this so called *overdispersion* and the solution described below will serve as an illustrative example of how to define custom families in **brms**.

The *beta-binomial* model is a generalization of the *binomial* model with an additional parameter to account for overdispersion. In the beta-binomial model, we do not predict the binomial probability \(p_i\) directly, but assume it to be beta distributed with hyperparameters \(\alpha > 0\) and \(\beta > 0\):

\[ p_i \sim \text{Beta}(\alpha_i, \beta_i) \]

The \(\alpha\) and \(\beta\) parameters are both hard to interprete and generally not recommended for use in regression models. Thus, we will apply a different parameterization with parameters \(\mu \in [0, 1]\) and \(\phi > 0\), which we will call \(\text{Beta2}\):

\[ \text{Beta2}(\mu, \phi) = \text{Beta}(\mu \phi, (1-\mu) \phi) \]

The parameters \(\mu\) and \(\phi\) specify the mean and precision parameter, respectively. By defining

\[ \mu_i = \frac{\exp(\eta_i)}{1 + \exp(\eta_i)} \]

we still predict the expected probability by means of our transformed linear predictor (as in the original binomial model), but account for potential overdispersion via the parameter \(\phi\).

The beta-binomial distribution is not natively supported in **brms** and so we will have to define it ourselves using the `custom_family`

function. This function requires the family’s name, the names of its parameters (`mu`

and `phi`

in our case), corresponding link functions (only applied if parameters are prediced), their theoretical lower and upper bounds (only applied if parameters are not predicted), information on whether the distribuion is discrete or continuous, and finally, whether additional non-parameter variables need to be passed to the distribution. For our beta-binomial example, this results in the following custom family:

```
beta_binomial2 <- custom_family(
"beta_binomial2", dpars = c("mu", "phi"),
links = c("logit", "log"), lb = c(NA, 0),
type = "int", vars = "vint1[n]"
)
```

The name `vint1`

for the variable containing the number of trials is not chosen arbitrarily as we will see below. Next, we have to provide the relevant **Stan** functions if the distribution is not defined in **Stan** itself. For the `beta_binomial2`

distribution, this is straight forward since the ordinal `beta_binomial`

distribution is already implemented.

```
stan_funs <- "
real beta_binomial2_lpmf(int y, real mu, real phi, int T) {
return beta_binomial_lpmf(y | T, mu * phi, (1 - mu) * phi);
}
int beta_binomial2_rng(real mu, real phi, int T) {
return beta_binomial_rng(T, mu * phi, (1 - mu) * phi);
}
"
```

For the model fitting, we will only need `beta_binomial2_lpmf`

, but `beta_binomial2_rng`

will come in handy when it comes to post-processing. We define:

To provide information about the number of trials (an integer variable), we are going to use the addition argument `vint()`

, which can only be used in custom families. Simiarily, if we needed to include additional vectors of real data, we would use `vreal()`

. Actually, for this particular example, we could more elegantly apply the addition argument `trials()`

instead of `vint()`

as in the basic binomial model. However, since the present vignette is ment to give a general overview of the topic, we will go with the more general method.

We now have all components together to fit our custom beta-binomial model:

```
fit2 <- brm(
incidence | vint(size) ~ period + (1|herd), data = cbpp,
family = beta_binomial2, stanvars = stanvars
)
```

The summary output reveals that the uncertainty in the coefficients of `period`

is somewhat larger than in the basic binomial model, which is the result of including the overdispersion parameter `phi`

in the model. Aparat from that, the results looks pretty similar.

```
Family: beta_binomial2
Links: mu = logit; phi = identity
Formula: incidence | vint(size) ~ period + (1 | herd)
Data: cbpp (Number of observations: 56)
Samples: 4 chains, each with iter = 2000; warmup = 1000; thin = 1;
total post-warmup samples = 4000
Group-Level Effects:
~herd (Number of levels: 15)
Estimate Est.Error l-95% CI u-95% CI Rhat Bulk_ESS Tail_ESS
sd(Intercept) 0.39 0.26 0.02 0.95 1.01 1058 1841
Population-Level Effects:
Estimate Est.Error l-95% CI u-95% CI Rhat Bulk_ESS Tail_ESS
Intercept -1.34 0.26 -1.85 -0.83 1.00 3342 2442
period2 -1.00 0.41 -1.84 -0.25 1.00 4283 2812
period3 -1.25 0.45 -2.20 -0.39 1.00 3837 2861
period4 -1.53 0.52 -2.66 -0.57 1.00 3586 2738
Family Specific Parameters:
Estimate Est.Error l-95% CI u-95% CI Rhat Bulk_ESS Tail_ESS
phi 17.59 18.01 5.48 55.87 1.00 1365 1103
Samples were drawn using sampling(NUTS). For each parameter, Bulk_ESS
and Tail_ESS are effective sample size measures, and Rhat is the potential
scale reduction factor on split chains (at convergence, Rhat = 1).
```

Some post-proecssing methods such as `summary`

or `plot`

work out of the box for custom family models. However, there are three particularily important methods, which require additional input by the user. These are `posterior_epred`

, `posterior_predict`

and `log_lik`

computing predicted mean values, predicted response values, and log-likelihood values, respectively. They are not only relevant for their own sake, but also provide the basis of many other post-processing methods. For instance, we may be interested in comparing the fit of the binomial model with that of the beta-binomial model by means of approximate leave-one-out cross-validation implemented in method `loo`

, which in turn requires `log_lik`

to be working.

The `log_lik`

function of a family should be named `log_lik_<family-name>`

and have the two arguments `i`

(indicating observations) and `prep`

. You don’t have to worry too much about how `prep`

is created (if you are interested, check out the `prepare_predictions`

function). Instead, all you need to know is that parameters are stored in slot `dpars`

and data are stored in slot `data`

. Generally, parameters take on the form of a \(S \times N\) matrix (with \(S =\) number of posterior samples and \(N =\) number of observations) if they are predicted (as is `mu`

in our example) and a vector of size \(N\) if the are not predicted (as is `phi`

).

We could define the complete log-likelihood function R directly, or we can expose the self-defined **Stan** functions and apply them. The latter approach is usually more convenient, but the former is more stable and the only option when implementing custom families in other R packages building upon **brms**. For the purpose of the present vignette, we will go with the latter approach

and define the required `log_lik`

functions with a few lines of code.

```
log_lik_beta_binomial2 <- function(i, prep) {
mu <- prep$dpars$mu[, i]
phi <- prep$dpars$phi
trials <- prep$data$vint1[i]
y <- prep$data$Y[i]
beta_binomial2_lpmf(y, mu, phi, trials)
}
```

With that being done, all of the post-processing methods requiring `log_lik`

will work as well. For instance, model comparison can simply be performed via

```
Output of model 'fit1':
Computed from 4000 by 56 log-likelihood matrix
Estimate SE
elpd_loo -99.9 10.1
p_loo 22.1 4.3
looic 199.9 20.3
------
Monte Carlo SE of elpd_loo is NA.
Pareto k diagnostic values:
Count Pct. Min. n_eff
(-Inf, 0.5] (good) 46 82.1% 305
(0.5, 0.7] (ok) 5 8.9% 404
(0.7, 1] (bad) 5 8.9% 46
(1, Inf) (very bad) 0 0.0% <NA>
See help('pareto-k-diagnostic') for details.
Output of model 'fit2':
Computed from 4000 by 56 log-likelihood matrix
Estimate SE
elpd_loo -94.9 8.3
p_loo 10.8 2.0
looic 189.9 16.5
------
Monte Carlo SE of elpd_loo is NA.
Pareto k diagnostic values:
Count Pct. Min. n_eff
(-Inf, 0.5] (good) 48 85.7% 775
(0.5, 0.7] (ok) 6 10.7% 255
(0.7, 1] (bad) 2 3.6% 28
(1, Inf) (very bad) 0 0.0% <NA>
See help('pareto-k-diagnostic') for details.
Model comparisons:
elpd_diff se_diff
fit2 0.0 0.0
fit1 -5.0 4.2
```

Since larger `ELPD`

values indicate better fit, we see that the beta-binomial model fits somewhat better, although the corresponding standard error reveals that the difference is not that substantial.

Next, we will define the function necessary for the `posterior_predict`

method:

```
posterior_predict_beta_binomial2 <- function(i, prep, ...) {
mu <- prep$dpars$mu[, i]
phi <- prep$dpars$phi
trials <- prep$data$vint1[i]
beta_binomial2_rng(mu, phi, trials)
}
```

The `posterior_predict`

function looks pretty similar to the corresponding `log_lik`

function, except that we are now creating random draws of the response instead of log-liklihood values. Again, we are using an exposed **Stan** function for convenience. Make sure to add a `...`

argument to your `posterior_predict`

function even if you are not using it, since some families require additional arguments. With `posterior_predict`

to be working, we can engage for instance in posterior-predictive checking:

When defining the `posterior_epred`

function, you have to keep in mind that it has only a `prep`

argument and should compute the mean response values for all observations at once. Since the mean of the beta-binomial distribution is \(\text{E}(y) = \mu T\) definition of the corresponding `posterior_epred`

function is not too complicated, but we need to get the dimension of parameters and data in line.

```
posterior_epred_beta_binomial2 <- function(prep) {
mu <- prep$dpars$mu
trials <- prep$data$vint1
trials <- matrix(trials, nrow = nrow(mu), ncol = ncol(mu), byrow = TRUE)
mu * trials
}
```

A post-processing method relying directly on `posterior_epred`

is `conditional_effects`

, which allows to visualize effects of predictors.

For ease of interpretation we have set `size`

to 1 so that the y-axis of the above plot indicates probabilities.

Family functions built natively into **brms** are saver to use and more convenient, as they require much less user input. If you think that your custom family is general enough to be useful to other users, please feel free to open an issue on GitHub so that we can discuss all the details. Provided that we agree it makes sense to implement your family natively in brms, the following steps are required (`foo`

is a placeholder for the family name):

- In
`family-lists.R`

, add function`.family_foo`

which should contain basic information about your family (you will find lots of examples for other families there). - In
`families.R`

, add family function`foo`

which should be a simple wrapper around`.brmsfamily`

. - In
`stan-likelihood.R`

, add function`stan_llh_foo`

which provides the likelihood of the family in Stan language. - If necessary, add self-defined Stan functions in separate files under
`inst/chunks`

. - Add functions
`posterior_predict_foo`

,`posterior_epred_foo`

and`log_lik_foo`

to`posterior_predict.R`

,`posterior_epred.R`

and`log_lik.R`

, respectively. - If necessary, add distribution functions to
`distributions.R`

.